Dysentery

Dysentery (UK: /ˈdɪsəntəri/,[1] US: /ˈdɪsəntɛri/),[2] historically known as the bloody flux,[3] is a type of gastroenteritis that results in bloody diarrhea.[4][5] Other symptoms may include fever, abdominal pain, and a feeling of incomplete defecation.[6][7][8] Complications may include dehydration.[9]

The cause of dysentery is usually the bacteria from genus Shigella, in which case it is known as shigellosis, or the amoeba Entamoeba histolytica; then it is called amoebiasis.[4] Other causes may include certain chemicals, other bacteria, other protozoa, or parasitic worms.[6] It may spread between people.[10] Risk factors include contamination of food and water with feces due to poor sanitation.[7] The underlying mechanism involves inflammation of the intestine, especially of the colon.[6]

Efforts to prevent dysentery include hand washing and food safety measures while traveling in countries of high risk.[10] While the condition generally resolves on its own within a week, drinking sufficient fluids such as oral rehydration solution is important.[10] Antibiotics such as azithromycin may be used to treat cases associated with travelling in the developing world.[8] While medications used to decrease diarrhea such as loperamide are not recommended on their own, they may be used together with antibiotics.[8][10]

Shigella results in about 165 million cases of diarrhea and 1.1 million deaths a year with nearly all cases in the developing world.[11] In areas with poor sanitation nearly half of cases of diarrhea are due to Entamoeba histolytica.[7] Entamoeba histolytica affects millions of people and results in more than 55,000 deaths a year.[12] It commonly occurs in less developed areas of Central and South America, Africa, and Asia.[12] Dysentery has been described at least since the time of Hippocrates.[13]

## Contents

* 1 Signs and symptoms
* 2 Cause
* 2.1 Amoebic dysentery
* 2.2 Bacillary dysentery
* 2.3 Other bacteria
* 3 Diagnosis
* 3.1 Physical exam
* 3.2 Stool and blood tests
* 4 Prevention
* 4.1 Vaccine
* 5 Treatment
* 6 Prognosis
* 7 Epidemiology
* 8 History
* 9 Notable cases
* 10 See also
* 11 References
* 11.1 Works cited

## Signs and symptoms

The most common form of dysentery is bacillary dysentery, which is typically a mild sickness, causing symptoms normally consisting of mild abdominal pains and frequent passage of loose stools or diarrhea. Symptoms normally present themselves after 1–3 days, and are usually no longer present after a week. The frequency of urges to defecate, the large volume of liquid feces ejected, and the presence of blood, mucus, or pus depends on the pathogen causing the disease. Temporary lactose intolerance can occur, as well. In some occasions, severe abdominal cramps, fever, shock, and delirium can all be symptoms.[6][14][15][16]

In extreme cases, people may pass more than one liter of fluid per hour. More often, individuals will complain of diarrhea with blood, accompanied by extreme abdominal pain, rectal pain and a low-grade fever. Rapid weight loss and muscle aches sometimes also accompany dysentery, while nausea and vomiting are rare.

On rare occasions, the amoebic parasite will invade the body through the bloodstream and spread beyond the intestines. In such cases, it may more seriously infect other organs such as the brain, lungs, and most commonly the liver.[17]

## Cause

Cross-section of diseased intestines. Colored lithograph c. 1843

Bacterial infections that cause bloody diarrhea are typically classified as being either invasive or toxogenic. Invasive species cause damage directly by invading into the mucosa. The toxogenic species do not invade, but cause cellular damage by secreting toxins, resulting in bloody diarrhea. This is also in contrast to toxins that cause watery diarrhea, which usually do not cause cellular damage, but rather they take over cellular machinery for a portion of life of the cell.[18]

Definitions of dysentery can vary by region and by medical specialty. The U. S. Centers for Disease Control and Prevention (CDC) limits its definition to "diarrhea with visible blood".[19] Others define the term more broadly.[20] These differences in definition must be taken into account when defining mechanisms. For example, using the CDC definition requires that intestinal tissue be so severely damaged that blood vessels have ruptured, allowing visible quantities of blood to be lost with defecation. Other definitions require less specific damage.

### Amoebic dysentery

Main article: Medicine:Amoebiasis

Amoebiasis, also known as amoebic dysentery, is caused by an infection from the amoeba Entamoeba histolytica,[21] which is found mainly in tropical areas.[22] Proper treatment of the underlying infection of amoebic dysentery is important; insufficiently treated amoebiasis can lie dormant for years and subsequently lead to severe, potentially fatal, complications. When amoebae inside the bowel of an infected person are ready to leave the body, they group together and form a shell that surrounds and protects them. This group of amoebae is known as a cyst, which is then passed out of the person's body in the feces and can survive outside the body. If hygiene standards are poor – for example, if the person does not dispose of the feces hygienically – then it can contaminate the surroundings, such as nearby food and water. If another person then eats or drinks food or water that has been contaminated with feces containing the cyst, that person will also become infected with the amoebae. Amoebic dysentery is particularly common in parts of the world where human feces are used as fertilizer.

### Bacillary dysentery

Dysentery may also be caused by shigellosis, an infection by bacteria of the genus Shigella, and is then known as bacillary dysentery (or Marlow syndrome). The term bacillary dysentery etymologically might seem to refer to any dysentery caused by any bacilliform bacteria, but its meaning is restricted by convention to Shigella dysentery.[citation needed]

### Other bacteria

Some strains of Escherichia coli cause bloody diarrhea. The typical culprits are enterohemorrhagic Escherichia coli, of which O157:H7 is the best known. These types of E. coli also make Shiga toxin.[23]

## Diagnosis

A diagnosis may be made by taking a history and doing a brief examination. Dysentery should not be confused with hematochezia, which is the passage of fresh blood through the anus, usually in or with stools.[24]

### Physical exam

The mouth, skin, and lips may appear dry due to dehydration. Lower abdominal tenderness may also be present.[17]

### Stool and blood tests

Cultures of stool samples are examined to identify the organism causing dysentery. Usually, several samples must be obtained due to the number of amoebae, which changes daily.[17] Blood tests can be used to measure abnormalities in the levels of essential minerals and salts.[17]

## Prevention

Efforts to prevent dysentery include hand washing and food safety measures while traveling in areas of high risk.[10]

### Vaccine

Although there is currently no vaccine that protects against Shigella infection, several are in development.[25][26] Vaccination may eventually become a part of the strategy to reduce the incidence and severity of diarrhea, particularly among children in low-resource settings. For example, Shigella is a longstanding World Health Organization (WHO) target for vaccine development, and sharp declines in age-specific diarrhea/dysentery attack rates for this pathogen indicate that natural immunity does develop following exposure; thus, vaccination to prevent this disease should be feasible. The development of vaccines against these types of infection has been hampered by technical constraints, insufficient support for coordination, and a lack of market forces for research and development. Most vaccine development efforts are taking place in the public sector or as research programs within biotechnology companies.

## Treatment

Dysentery is managed by maintaining fluids using oral rehydration therapy.[10] If this treatment cannot be adequately maintained due to vomiting or the profuseness of diarrhea, hospital admission may be required for intravenous fluid replacement. In ideal situations, no antimicrobial therapy should be administered until microbiological microscopy and culture studies have established the specific infection involved. When laboratory services are not available, it may be necessary to administer a combination of drugs, including an amoebicidal drug to kill the parasite, and an antibiotic to treat any associated bacterial infection.[citation needed] Laudanum (Deodorized Tincture of Opium) may be used for severe pain and to combat severe diarrhea.[citation needed]

If shigellosis is suspected and it is not too severe, letting it run its course may be reasonable – usually less than a week. If the case is severe, antibiotics such as ciprofloxacin or TMP-SMX may be useful. However, many strains of Shigella are becoming resistant to common antibiotics, and effective medications are often in short supply in developing countries. If necessary, a doctor may have to reserve antibiotics for those at highest risk for death, including young children, people over 50, and anyone suffering from dehydration or malnutrition.[citation needed]

Amoebic dysentery is often treated with two antimicrobial drugs such as metronidazole and paromomycin or iodoquinol.[27]

## Prognosis

With correct treatment, most cases of amoebic and bacterial dysentery subside within 10 days, and most individuals achieve a full recovery within two to four weeks after beginning proper treatment. If the disease is left untreated, the prognosis varies with the immune status of the individual patient and the severity of disease. Extreme dehydration can delay recovery and significantly raises the risk for serious complications including death.[28]

## Epidemiology

Insufficient data exists, but Shigella is estimated to have caused the death of 34,000 children under the age of five in 2013, and 40,000 deaths in people over five years of age.[25] Amoebiasis infects over 50 million people each year, of whom 50,000 die (one per thousand).[29]

## History

Shigella evolved with the human expansion out of Africa 50,000 to 200,000 years ago.[30]

The seed, leaves, and bark of the kapok tree have been used in traditional medicines by indigenous peoples of the rainforest regions in the Americas, west-central Africa, and Southeast Asia in the treatment of this disease.[31][32][33]

In 1915, Australian bacteriologist Fannie Eleanor Williams was serving as a medic in Greece with the Australian Imperial Force, receiving casualties directly from Gallipoli. In Gallipoli, dysentery was severely affecting soldiers and causing significant loss of manpower. Williams carried out serological investigations into dysentery, co-authoring several groundbreaking papers with Sir Charles Martin, director of the Lister Institute.[34] The result of their work into dysentery was increased demand for specific diagnostics and curative sera.[35]

Bacillus subtilis was marketed throughout America and Europe from 1946 as an immunostimulatory aid in the treatment of gut and urinary tract diseases such as rotavirus and Shigella,[36] but declined in popularity after the introduction of consumer antibiotics.

## Notable cases

A Red Army soldier dies of dysentery after eating unwashed vegetables. This is a common way of contracting dysentery. From a health advisory pamphlet given to soldiers.

* 580: Childesinda, son of Chilperic I, Frankish king, died of dysentery as a child
* 580: Austregilde, Frankish queen, died of dysentery. According to Gregory of Tours she blamed her doctors for her death and asked her husband, King Guntram, to have the doctors executed after she died, which he did.[37]
* 642: Cyrus of Alexandria, Greek Orthodox Patriarch of Alexandria and governor of Egypt, died of dysentery on 21 March 642.
* 685: Constantine IV, the Byzantine emperor, died of dysentery on September 685.
* 1183: Henry the Young King died of dysentery at the castle of Martel on 11 June 1183.
* 1216: John, King of England died of dysentery at Newark Castle on 19 October 1216.[38]
* 1270: Louis IX of France died of dysentery in Tunis while commanding his troops for the Eighth Crusade on 25 August 1270.
* 1307: Edward I of England caught dysentery on his way to the Scottish border and died in his servants' arms on 7 July 1307.
* 1322: Philip V of France died of dysentery at the Abbey of Longchamp (site of the present hippodrome in the Bois de Boulogne) in Paris while visiting his daughter, Blanche, who had taken her vows as a nun there in 1322. He died on 3 January 1322.
* 1376: Edward the Black Prince, son of Edward III of England and heir to the English throne. Died of apparent dysentery in June, after a months-long period of illness during which he predicted his own imminent death, in his 46th year.
* 1422: King Henry V of England died suddenly on 31 August 1422 at the Château de Vincennes, apparently from dysentery,[39] which he had contracted during the siege of Meaux. He was 35 years old and had reigned for nine years.
* 1536: Erasmus, Dutch renaissance humanist and theologian. At Basel.[40]
* 1596: Sir Francis Drake, vice admiral, died of dysentery on 28 January 1596 whilst anchored off the coast of Portobelo.[41]
* 1605: Akbar, ruler of the Mughal Empire of South Asia, died of dysentery. On 3 October 1605, he fell ill with an attack of dysentery, from which he never recovered. He is believed to have died on or about 27 October 1605, after which his body was buried in a mausoleum in Agra, present-day India.[42]
* 1638: Sibylla Schwarz, a young German poet of the baroque era, died of dysentery during the Thirty Year's War in Pommerania at the age of 17.
* 1675: Jacques Marquette died of dysentery on his way north from what is today Chicago, traveling to the mission where he intended to spend the rest of his life.[43]
* 1676: Nathaniel Bacon died of dysentery after taking control of Virginia following Bacon's Rebellion. He is believed to have died in October 1676, allowing Virginia's ruling elite to regain control.[44]
* 1680: Shivaji, founder and ruler of the Maratha Empire of South Asia, died of dysentery on 3 April 1680. In 1680, Shivaji fell ill with fever and dysentery, dying around 3–5 April 1680 at the age of 52 on the eve of Hanuman Jayanti. He was cremated at Raigad Fort, where his Samadhi is built in Mahad, Raigad district of Maharashtra, India.[45][46]
* 1827: Queen Nandi kaBhebhe, (mother of Shaka Zulu) died of dysentery on 10 October 1827.[47]Allied prisoners of war with dysentery in a Japanese camp in Thailand in 1943
* 1873: The explorer David Livingstone died of dysentery on 1 May 1873.[48]
* 1885: Alfonso XII King of Spain from 29 December 1874 to 25 November 1885 died of dysentery at the age of 27
* 1896: Phan Đình Phùng, a Vietnamese revolutionary who led rebel armies against French colonial forces in Vietnam, died of dysentery as the French surrounded his forces on 21 January 1896.[49]
* 1910: Luo Yixiu, first wife of Mao Zedong, died of dysentery on 11 February 1910. She was 20 years old.({{{1}}}, {{{2}}})
* 1912: Historian Arnold J. Toynbee contracted dysentery on April 26, 1912, from contaminated water in the Peloponnese; the malady led to his being excused from military service in World War I.[50]
* 1930: The French explorer and writer Michel Vieuchange died of dysentery in Agadir on 30 November 1930, on his return from the "forbidden city" of Smara. He was nursed by his brother, Doctor Jean Vieuchange, who was unable to save him. The notebooks and photographs, edited by Jean Vieuchange, went on to become bestsellers.[51][52]
* 1942: The Selarang Barracks incident in the summer of 1942 during World War II involved the forced crowding of 17,000 Anglo-Australian prisoners-of-war (POWs) by their Japanese captors in the areas around the barracks square for nearly five days with little water and no sanitation after the Selarang Barracks POWs refused to sign a pledge not to escape. The incident ended with the surrender of the Australian commanders due to the spreading of dysentery among their men.[53]

## See also

* Cholera

## References

1. ↑ "dysentery". dysentery. Oxford University Press. n.d.. https://web.archive.org/www.lexico.com/definition/dysentery. Retrieved 2021-08-31.
2. ↑ "Dysentery". Merriam-Webster Dictionary. https://www.merriam-webster.com/dictionary/dysentery. Retrieved 2022-04-24.
3. ↑ bloody flux (3rd ed.), Oxford University Press, September 2005, http://www.oed.com/view/Entry/302050 (Subscription or UK public library membership required.)
4. ↑ 4.0 4.1 "Dysentery". https://www.who.int/topics/dysentery/en/.
5. ↑ "Controlling the Spread of Infections in Evacuation Centers |Health and Safety Concerns". U.S. Centers for Disease Control and Prevention. https://www.cdc.gov/disasters/disease/infectevac.html.
6. ↑ 6.0 6.1 6.2 6.3 "Dysentery" at Dorland's Medical Dictionary
7. ↑ 7.0 7.1 7.2 "Amoebic dysentery". BMJ Clinical Evidence 2013. August 2013. PMID 23991750.
8. ↑ 8.0 8.1 8.2 "Antibiotic Therapy for Acute Watery Diarrhea and Dysentery". Military Medicine 182 (S2): 17–25. September 2017. doi:10.7205/MILMED-D-17-00068. PMID 28885920.
9. ↑ "WHO EMRO | Dysentery | Health topics". http://www.emro.who.int/health-topics/dysentery/index.html.
10. ↑ 10.0 10.1 10.2 10.3 10.4 10.5 "Dysentery" (in en). 18 October 2017. https://www.nhs.uk/conditions/dysentery/.
11. ↑ "Dysentery (Shigellosis)". November 2016. p. 2. https://www.who.int/selection_medicines/committees/expert/21/applications/s6_paed_antibiotics_appendix5_dysentery.pdf.
12. ↑ 12.0 12.1 "A Review of the Global Burden, New Diagnostics, and Current Therapeutics for Amebiasis". Open Forum Infectious Diseases 5 (7). July 2018. doi:10.1093/ofid/ofy161. PMID 30046644.
13. ↑ (in en) Tapeworms, Lice, and Prions: A compendium of unpleasant infections. OUP Oxford. 2013. p. PT517. ISBN 978-0-8493-0072-1. https://books.google.com/books?id=KpxKAgAAQBAJ&pg=PT517.
14. ↑ "Interventions in diarrheas of infants and young children". Journal of the American Veterinary Medical Association 173 (5 Pt 2): 649–53. September 1978. doi:10.2460/javma.1978.173.05.649. PMID 359524.
15. ↑ "Acute diarrhea in children". Pediatrics in Review 11 (1): 6–13. July 1989. doi:10.1542/pir.11-1-6. PMID 2664748.
16. ↑ "Dysentery symptoms". http://www.nhs.uk/Conditions/Dysentery/Pages/Symptoms.aspx.
17. ↑ 17.0 17.1 17.2 17.3 "Dysentery-Diagnosis". http://www.mdguidelines.com/dysentery/diagnosis.
18. ↑ Boards and Beyond: Infectious Disease: A Companion Book to the Boards and Beyond Website (Version 9-26-2016 ed.). CreateSpace Independent Publishing Platform. 2016. ISBN 978-1-5237-0935-9.
19. ↑ "Laboratory Methods for the Diagnosis of Epidemic Dysentery and Cholera". WHO/CDS/CSR/EDC/99.8. Centers for Disease Control and Prevention. Atlanta, Georgia 1999. https://www.cdc.gov/ncidod/dbmd/diseaseinfo/cholera/complete.pdf.
20. ↑ "Dysentery". TheFreeDictionary's Medical dictionary. http://medical-dictionary.thefreedictionary.com/dysentery.
21. ↑ WHO (1969). "Amoebiasis. Report of a WHO Expert Committee". WHO Technical Report Series 421: 1–52. PMID 4978968.
22. ↑ Amebic+Dysentery at the US National Library of Medicine Medical Subject Headings (MeSH)
23. ↑ Phillips, A; Navabpour, S; Hicks, S; Dougan, G; Wallis, T; Frankel, G (2000). "Enterohaemorrhagic Escherichia coli O157:H7 target Peyer's patches in humans and cause attaching/effacing lesions in both human and bovine intestine". Gut 47 (3): 377–381. doi:10.1136/gut.47.3.377. PMID 10940275.
24. ↑ Wilson, I. Dodd (1990), Walker, H. Kenneth; Hall, W. Dallas; Hurst, J. Willis, eds., "Hematemesis, Melena, and Hematochezia", Clinical Methods: The History, Physical, and Laboratory Examinations (Boston: Butterworths), ISBN 978-0-409-90077-4, PMID 21250251, https://www.ncbi.nlm.nih.gov/books/NBK411/, retrieved 2024-10-28
25. ↑ 25.0 25.1 "Status of vaccine research and development for Shigella". Vaccine 34 (26): 2887–2894. June 2016. doi:10.1016/j.vaccine.2016.02.075. PMID 26979135.
26. ↑ "WHO vaccine pipeline tracker" (in en-GB). http://who.int/immunization/research/vaccine_pipeline_tracker_spreadsheet/en/.
27. ↑ "Chapter 3 Infectious Diseases Related To Travel". August 1, 2013. http://wwwnc.cdc.gov/travel/yellowbook/2014/chapter-3-infectious-diseases-related-to-travel/amebiasis.
28. ↑ mdguidelines.com. "Dysentery-Prognosis". http://www.mdguidelines.com/dysentery/prognosis.
29. ↑ Encyclopedia of Pestilence, Pandemics, and Plagues: A-M. ABC-CLIO. 2008. pp. 175–176. ISBN 978-0-313-34102-1. https://books.google.com/books?id=5Pvi-ksuKFIC&pg=PA175.
30. ↑ Pupo, Gulietta M.; Lan, Ruiting; Reeves, Peter R. (2000-08-22). "Multiple independent origins of Shigella clones ofEscherichia coliand convergent evolution of many of their characteristics". Proceedings of the National Academy of Sciences 97 (19): 10567–10572. doi:10.1073/pnas.180094797. ISSN 0027-8424. PMID 10954745. Bibcode: 2000PNAS...9710567P.
31. ↑ "Kapok Tree". Blue Planet and Biomoes. http://www.blueplanetbiomes.org/kapok.htm.
32. ↑ "Ceiba pentandra". Human Uses and Cultural Importance. http://www.zoo.org/page.aspx?pid=1954.
33. ↑ "Kapok Emergent Tree Of Tropical Rain Forest Used To Treat Asthma Dysentery Fever Kidney Diseases". http://www.encyclocenter.com/Kapok-Emergent-24736.html.
34. ↑ Digital, Carter. "Fannie Williams" (in en). https://discovery.wehi.edu.au/timeline/fannie-williams.
35. ↑ Kirsty Harris. "Fannie Eleanor Williams: Bacteriologist and Serologist". Seizing the Initiative: Australian Women Leaders in Politics, Workplaces and Communities.. http://www.womenaustralia.info/leaders/sti/pdfs/10_Harris.pdf.
36. ↑ "The use of Bacillus subtilis as an antidiarrhoeal microorganism". Bollettino Chimico Farmaceutico 133 (1): 3–18. January 1994. PMID 8166962.
37. ↑ Gregory of Tours. A History of the Franks, Pantianos Classics, 1916
38. ↑ King John. London: Methuen. 1991. p. 253. ISBN 978-0-413-45520-8. https://books.google.com/books?id=Vt5HPgAACAAJ&pg=PA253.
39. ↑ "BBC – History – Henry V". https://www.bbc.co.uk/history/historic_figures/henry_v_king.shtml.
40. ↑ Herbermann, Charles, ed (1913). "Desiderius Erasmus". Catholic Encyclopedia. New York: Robert Appleton Company.
41. ↑ "BBC – History – Sir Francis Drake". https://www.bbc.co.uk/history/historic_figures/drake_francis.shtml.
42. ↑ Majumdar 1984, pp. 168–169
43. ↑ "Louis Jolliet (1645-1700) and Jacques Marquette (1637-1675)". Discoverers Web. http://www.win.tue.nl/~engels/discovery/jolmar.html.
44. ↑ Give Me Liberty! An American History (brief ed.). New York; London: W. W. Norton and Company. 2012. ISBN 978-0-393-92032-1.
45. ↑ Shivaji and His Times (Second ed.). London: Longmans, Green and Co.. 1920. https://archive.org/details/shivajihistimes00sarkrich.
46. ↑ Sarkar, Shivaji and His Times 1920, p. 382.
47. ↑ The washing of the spears: a history of the rise of the Zulu nation under Shaka and its fall in the Zulu War of 1879 (1st ed.). New York: Da Capo Press. 1998. ISBN 978-0-306-80866-1.
48. ↑ The Last Journals of David Livingstone, in Central Africa, from 1865 to His Death: Continued by a Narrative of His Last Moments and Sufferings, Obtained from His Faithful Servants Chuma and Susi; in Two Volumes. J. Murray. 1874. https://books.google.com/books?id=S_dgAAAAcAAJ.
49. ↑ Vietnamese anticolonialism, 1885–1925. Berkeley, California: University of California. 1970. p. 68. ISBN 978-0-520-01813-6. https://books.google.com/books?id=lu_TB0X8B4YC&pg=PA68.
50. ↑ William H. McNeill, Arnold Toynbee: A Life (Oxford University Press, 1989), pp. 40, 45, & 67-69.
51. ↑ (in fr) L'ultime désert: vie et mort de Michel Vieuchange. Paris: Phébus. 2004. pp. 29, 245–249 & 253. ISBN 978-2-85940-997-5.
52. ↑ Smara: The Forbidden City. Fletcher Allen, Edgar (translation); Vieuchange, Jean (editor; introduction, notes, postscript); Claudel, Paul (preface). (Reprint ed.). New York: Ecco. 1988. ISBN 978-0-88001-146-4.
53. ↑ The Battle For Singapore—The True Story of the Greatest Catastrophe of World War II. United Kingdom: Portraits Books. 2005. pp. 389–390. ISBN 978-0-7499-5085-9.

### Works cited

* Chang, Jung; Halliday, Jon (2005). Mao: The Unknown Story. London: Jonathan Cape. ISBN 978-0-224-07126-0.
* Lü, Chun 吕春 (2009). "Six Women who Influenced Mao Zedong 影响毛泽东一生的六位女性". http://dangshi.people.com.cn/GB/144956/11272977.html.
* Majumdar, R.C. (1984). The Mughul Empire. Bombay: Bharatiya Vidya Bhavan.
* Pantsov, Alexander V.; Levine, Steven I. (2012). Mao: The Real Story. New York and London: Simon & Schuster. ISBN 978-1-4516-5447-9.

| Wikimedia Commons has media related to Dysentery.

|

Classification|

* ICD-10: A09.0, A03.9, A06.0, A07.9
* ICD-9-CM: 004, 007.9, 009.0
* MeSH: D004403

|

* v
* t
* e

Infectious diseases – Parasitic disease: protozoan infection: Excavata (A06–A07, B55–B57, 007, 085–086)

Discicristata|

| Trypanosomatida|

| Trypanosomiasis|

* T. brucei
* African trypanosomiasis
* T. cruzi
* Chagas disease

|

Leishmaniasis|

* Leishmania major / L. mexicana / L. aethiopica / L. tropica
* Cutaneous leishmaniasis
* L. braziliensis
* Mucocutaneous leishmaniasis
* L. donovani / infantum
* Visceral leishmaniasis

Schizopyrenida|

* Naegleria fowleri
* Primary amoebic meningoencephalitis

Trichozoa|

| Diplomonadida|

* Giardia lamblia (Giardiasis)

|

Trichomonadida|

* Trichomonas vaginalis
* Trichomoniasis
* Dientamoeba fragilis
* Dientamoebiasis

* v
* t
* e

* Infectious diseases
* Bacterial disease: ProteobacterialG−]]
* primarily A00–A79, 001–041, 080–109

| Rickettsiales|

| Rickettsiaceae/
(Rickettsioses)|

| Typhus|

* Rickettsia typhi
* Murine typhus
* Rickettsia prowazekii
* Epidemic typhus, Brill–Zinsser disease, Flying squirrel typhus

|

Spotted
fever|

| Tick-borne|

* Rickettsia rickettsii
* Rocky Mountain spotted fever
* Rickettsia conorii
* Boutonneuse fever
* Rickettsia japonica
* Japanese spotted fever
* Rickettsia sibirica
* North Asian tick typhus
* Rickettsia australis
* Queensland tick typhus
* Rickettsia honei
* Flinders Island spotted fever
* Rickettsia africae
* African tick bite fever
* Rickettsia parkeri
* American tick bite fever
* Rickettsia aeschlimannii
* Rickettsia aeschlimannii infection

|

Mite-borne|

* Rickettsia akari
* Rickettsialpox
* Orientia tsutsugamushi
* Scrub typhus

Flea-borne|

* Rickettsia felis
* Flea-borne spotted fever

Anaplasmataceae|

* Ehrlichiosis: Anaplasma phagocytophilum
* Human granulocytic anaplasmosis, Anaplasmosis
* Ehrlichia chaffeensis
* Human monocytotropic ehrlichiosis
* Ehrlichia ewingii
* Ehrlichiosis ewingii infection

Rhizobiales|

| Brucellaceae|

* Brucella abortus
* Brucellosis

|

Bartonellaceae|

* Bartonellosis: Bartonella henselae
* Cat-scratch disease
* Bartonella quintana
* Trench fever
* Either B. henselae or B. quintana
* Bacillary angiomatosis
* Bartonella bacilliformis
* Carrion's disease, Verruga peruana

| Neisseriales|

| M+|

* Neisseria meningitidis/meningococcus
* Meningococcal disease, Waterhouse–Friderichsen syndrome]], Meningococcal septicaemia

|

M−|

* Neisseria gonorrhoeae/gonococcus
* Gonorrhea

ungrouped:|

* Eikenella corrodens/Kingella kingae
* HACEK
* Chromobacterium violaceum
* Chromobacteriosis infection

Burkholderiales|

* Burkholderia pseudomallei
* Melioidosis
* Burkholderia mallei
* Glanders
* Burkholderia cepacia complex
* Bordetella pertussis/Bordetella parapertussis
* Pertussis

| Enterobacteriales
(OX−)|

| Lac+|

* Klebsiella pneumoniae
* Rhinoscleroma, Pneumonia
* Klebsiella granulomatis
* Granuloma inguinale
* Klebsiella oxytoca

* Escherichia coli: Enterotoxigenic
* Enteroinvasive
* Enterohemorrhagic
* O157:H7
* O104:H4
* Hemolytic-uremic syndrome

* Enterobacter aerogenes/Enterobacter cloacae

|

Slow/weak|

* Serratia marcescens
* Serratia infection
* Citrobacter koseri/Citrobacter freundii

Lac−|

| H2S+|

* Salmonella enterica
* Typhoid fever, Paratyphoid fever, Salmonellosis

|

H2S−|

* Shigella dysenteriae/sonnei/flexneri]]/boydii
* Shigellosis, Bacillary dysentery
* Proteus mirabilis/Proteus vulgaris
* Yersinia pestis
* Plague/Bubonic plague
* Yersinia enterocolitica
* Yersiniosis
* Yersinia pseudotuberculosis
* Far East scarlet-like fever

Pasteurellales|

| Haemophilus:|

* H. influenzae
* Haemophilus meningitis
* Brazilian purpuric fever
* H. ducreyi
* Chancroid
* H. parainfluenzae
* HACEK

|

Pasteurella multocida|

* Pasteurellosis
* Actinobacillus
* Actinobacillosis

Aggregatibacter actinomycetemcomitans|

* HACEK

Legionellales|

* Legionella pneumophila/Legionella longbeachae
* Legionnaires' disease
* Coxiella burnetii
* Q fever

Thiotrichales|

* Francisella tularensis
* Tularemia

Vibrionaceae|

* Vibrio cholerae
* Cholera
* Vibrio vulnificus
* Vibrio parahaemolyticus
* Vibrio alginolyticus
* Plesiomonas shigelloides

Pseudomonadales|

* Pseudomonas aeruginosa
* Pseudomonas infection
* Moraxella catarrhalis
* Acinetobacter baumannii

Xanthomonadaceae|

* Stenotrophomonas maltophilia

Cardiobacteriaceae|

* Cardiobacterium hominis
* HACEK

Aeromonadales|

* Aeromonas hydrophila/Aeromonas veronii
* Aeromonas infection

| Campylobacterales|

* Campylobacter jejuni
* Campylobacteriosis, Guillain–Barré syndrome]]
* Helicobacter pylori
* Peptic ulcer, MALT lymphoma, Gastric cancer
* Helicobacter cinaedi
* Helicobacter cellulitis

|

* v
* t
* e

Diseases of the digestive system (primarily K20–K93, 530–579)

Upper GI tract|

| Esophagus|

* Esophagitis
* Candidal
* Eosinophilic
* Herpetiform
* Rupture
* Boerhaave syndrome
* Mallory–Weiss syndrome
* UES
* Zenker's diverticulum
* LES
* Barrett's esophagus
* Esophageal motility disorder
* Nutcracker esophagus
* Achalasia
* Diffuse esophageal spasm
* Gastroesophageal reflux disease (GERD)
* Laryngopharyngeal reflux (LPR)
* Esophageal stricture
* Megaesophagus

|

Stomach|

* Gastritis
* Atrophic
* Ménétrier's disease
* Gastroenteritis
* Peptic (gastric) ulcer
* Cushing ulcer
* Dieulafoy's lesion
* Dyspepsia
* Pyloric stenosis
* Achlorhydria
* Gastroparesis
* Gastroptosis
* Portal hypertensive gastropathy
* Gastric antral vascular ectasia
* Gastric dumping syndrome
* Gastric volvulus
* Buried bumper syndrome

Lower GI tract:
Intestinal/
Enteropathy|

| Small intestine
(Duodenum/Jejunum/Ileum)|

* Enteritis
* Duodenitis
* Jejunitis
* Ileitis
* Peptic (duodenal) ulcer
* Curling's ulcer
* Malabsorption: Coeliac
* Tropical sprue
* Blind loop syndrome
* Small bowel bacterial overgrowth syndrome
* Whipple's
* Short bowel syndrome
* Steatorrhea
* Milroy disease
* Bile acid malabsorption

|

Large intestine
(Appendix/Colon)|

* Appendicitis
* Colitis
* Pseudomembranous
* Ulcerative
* Ischemic
* Microscopic
* Collagenous
* Lymphocytic
* Functional colonic disease
* IBS
* Intestinal pseudoobstruction / Ogilvie syndrome
* Megacolon / Toxic megacolon
* Diverticulitis/Diverticulosis

Large and/or small|

* Enterocolitis
* Necrotizing
* Gastroenterocolitis
* IBD
* Crohn's disease
* Vascular: Abdominal angina
* Mesenteric ischemia
* Angiodysplasia
* Bowel obstruction: Ileus
* Intussusception
* Volvulus
* Fecal impaction
* Constipation
* Diarrhea
* Infectious
* Intestinal adhesions

Rectum|

* Proctitis
* Radiation proctitis
* Proctalgia fugax
* Rectal prolapse
* Anismus

Anal canal|

* Anal fissure/Anal fistula
* Anal abscess
* Hemorrhoid
* Anal dysplasia
* Pruritus ani

GI bleeding/BIS|

* Upper
* Hematemesis
* Melena
* Lower
* Hematochezia

Accessory|

| Liver|

* Hepatitis
* Viral hepatitis
* Autoimmune hepatitis
* Alcoholic hepatitis
* Cirrhosis
* PBC
* Fatty liver
* NASH
* Vascular
* Budd–Chiari syndrome
* Hepatic veno-occlusive disease
* Portal hypertension
* Nutmeg liver
* Alcoholic liver disease
* Liver failure
* Hepatic encephalopathy
* Acute liver failure
* Liver abscess
* Pyogenic
* Amoebic
* Hepatorenal syndrome
* Peliosis hepatis
* Metabolic disorders
* Wilson's disease
* Hemochromatosis

|

Gallbladder|

* Cholecystitis
* Gallstones/Cholecystolithiasis
* Cholesterolosis
* Adenomyomatosis
* Postcholecystectomy syndrome
* Porcelain gallbladder

Bile duct/
Other biliary tree|

* Cholangitis
* Primary sclerosing cholangitis
* Secondary sclerosing cholangitis
* Ascending
* Cholestasis/Mirizzi's syndrome
* Biliary fistula
* Haemobilia
* Gallstones/Cholelithiasis

* Common bile duct
* Choledocholithiasis
* Biliary dyskinesia
* Sphincter of Oddi dysfunction

Pancreatic|

* Pancreatitis
* Acute
* Chronic
* Hereditary
* Pancreatic abscess
* Pancreatic pseudocyst
* Exocrine pancreatic insufficiency
* Pancreatic fistula

Abdominopelvic|

| Hernia|

* Diaphragmatic
* Congenital
* Hiatus

* Inguinal
* Indirect
* Direct
* Umbilical
* Femoral
* Obturator
* Spigelian

* Lumbar
* Petit's
* Grynfeltt-Lesshaft

* Undefined location
* Incisional
* Internal hernia
* Richter's

|

Peritoneal|

* Peritonitis
* Spontaneous bacterial peritonitis
* Hemoperitoneum
* Pneumoperitoneum

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Original source: https://en.wikipedia.org/wiki/Dysentery. Read more

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