Ventricular enlargement not entirely attributable to cerebral atrophy or congenital enlargement (Evans index >0.3). No macroscopic obstruction to CSF flow. Enlargement of the temporal horns of the lateral ventricles not entirely attributable to hippocampus atrophy. Callosal angle of 40º or greater. Evidence of altered brain water content, including periventricular signal changes on CT and MRI not attributable to microvascular ischemic changes or demyelination. An aqueductal or fourth ventricular flow void.
Ventricular enlargement not entirely attributable to cerebral atrophy or congenital enlargement (Evans index >0.3).
No macroscopic obstruction to CSF flow.
At least one of the following supportive features:
Enlargement of the temporal horns of the lateral ventricles not entirely attributable to hippocampus atrophy;
Callosal angle of 40º or greater;
Evidence of altered brain water content, including periventricular signal changes on CT and MRI not attributable to microvascular ischemic changes or demyelination;
↑Toma AK, Holl E, Kitchen ND, Watkins LD (April 2011). "Evans' index revisited: the need for an alternative in normal pressure hydrocephalus". Neurosurgery. 68 (4): 939–44. doi:10.1227/NEU.0b013e318208f5e0. PMID21221031.
↑Mori E, Ishikawa M, Kato T, Kazui H, Miyake H, Miyajima M, Nakajima M, Hashimoto M, Kuriyama N, Tokuda T, Ishii K, Kaijima M, Hirata Y, Saito M, Arai H (2012). "Guidelines for management of idiopathic normal pressure hydrocephalus: second edition". Neurol. Med. Chir. (Tokyo). 52 (11): 775–809. PMID23183074.